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CL-Only Protocol
CARDIAC ANES
Vscan Air CL Curvilinear — Subcostal-First Approach • One Window, Both Answers
CL-Only Protocol
One Window, Both Answers, Under 30 Seconds
Start subxiphoid. Don't move. Get EF + volume status from the same position.
THE CL PROTOCOL CURVILINEAR OPTIMIZED
1
Subcostal 4-Chamber → Eyeball EF ⚡ ~10 sec
Probe flat under xiphoid, indicator to patient's LEFT, aim toward left shoulder. You'll see all 4 chambers. Eyeball: does the LV squeeze?
↔ Supine⏸ End-ExpCL StrengthAll States
Walls squeeze, cavity shrinks = normal Sluggish walls = reduced Barely moves, globular = severe
↓Don't move — just rotate the probe
2
IVC → Volume Status ⚡ ~15 sec
From subcostal 4C, flatten the probe toward skin and rotate clockwise ~90° to pick up the IVC running into RA. Measure max/min diameter.
↔ SupineSpontaneous → cIVCPPV → dIVC
↓Only if Step 1 is unclear
3
Fallback: PLAX Attempt → EPSS / FS 🔢 ~30+ sec
Only if subcostal EF assessment is equivocal (mild reduction you can't categorize). Move to parasternal, tilt probe on edge. This is your backup, not your primary.
↔ Supine + left tilt⏸ End-Exp⚠ CL Limitation
Step 1 — Probe Placement
Subcostal 4-Chamber View
Probe Position
Anatomy Cross-Section
Sub 4C — Labeled
Sub 4C — Self-Test
Step 1 — Assessment
Eyeball EF — Visual Estimation
Studies show trained providers categorize EF within ±5% of formal Simpson's. You're triaging, not writing an echo report. Ask: "How much does the LV cavity shrink from diastole to systole?"
Walls nearly motionless Globular, dilated LV Almost no size change
What to Look For — Quick Checklist
💪
Wall thickening: Do the walls get THICKER during systole? Normal myocardium thickens >40% — you can see this.
📏
Cavity size change: How much does the LV cavity shrink? Dramatic = normal. Minimal = bad.
🚪
MV excursion: Does the mitral valve open wide and briskly? Poor opening = poor filling OR poor function.
🔵
LV shape: Normal = bullet-shaped. Severe ↓ = round/globular. Globular LV = bad sign.
⚖️
RV vs LV size: RV should be smaller than LV. RV ≥ LV = RV dilation (PE? RV failure?)
⚠ Eyeball EF Limitations
Eyeball EF is a categorical estimate (normal / mild / mod / severe) — not a precise number. It's excellent for triaging but don't chart "EF 42% by eyeball." Chart "grossly normal" or "appears moderately reduced." If you need a number, fall back to PLAX for EPSS/FS. Under GA, volatile agents ↓ contractility 5–10% — account for this visually.
Step 2 — Volume Status
IVC — Slide From Subcostal 4C
You're already in position. Flatten the probe and rotate clockwise ~90° to pick up the IVC. Measure 2–3cm from RA-IVC junction.
IVC Probe Position
IVC — Labeled (HV, IVC, RA)
IVC — Self-Test
⚠ Pitfall: Aorta (not IVC!)
2D: End-Expiration — IVC 1.24cm (MAX)
2D: End-Inspiration — IVC 0.76cm (MIN)
M-Mode: End-Exp — IVC 2.08cm
M-Mode: End-Insp — IVC 1.78cm
IVC → CVP Reference
⚠ Don't Confuse IVC with Aorta
IVC vs Aorta Identification
IVC: Thin-walled, compressible, collapses with sniff, connects to RA, hepatic veins drain into it
Aorta: Thick-walled, pulsatile, NOT compressible, vertebral shadowing deep to it, celiac trunk branches off
IVC is to patient's RIGHT of the spine, aorta is LEFT
If you see vertebral shadowing and a thick bright wall — you're on the aorta, slide RIGHT
If you see hepatic veins draining in — you're on the IVC ✓
Transition from Sub4C → IVC
The Slide Maneuver
Start: You're in subcostal 4C (indicator → patient's left)
Flatten the probe even more toward skin
Rotate clockwise ~90° so indicator points cephalad (toward head)
Slide slightly to patient's right of midline
You should see the IVC running longitudinally into the RA
Measure 2–3cm from the RA-IVC junction
IVC Decision
BREATHING MODE?
SPONTANEOUS
cIVC ÷ IVC max (exp) Max at end-exp Min during sniff
cIVC >12% may predict fluid responsiveness in spontaneously breathing patients
Positive Pressure Ventilation
dIVC — Distensibility Calculator
↔ SupinePPV OnlyPassive, Vt ≥8
dIVC (%) = (IVC max − IVC min) / IVC min × 100
Denominator = MIN — physiology is reversed under PPV
Enter IVC Diameters (On Vent)
IVC maxcm (during insp)
IVC mincm (during exp)
— %
Distensibility
dIVC
Fluid Responsive?
Action
> 18%
YES
Fluid bolus
12–18%
GRAY ZONE
PLR, PPV, SVV
< 12%
NO
Pressors / inotropes
⚠ dIVC Invalid If
Spontaneous effort • Vt <8 mL/kg • Open chest • Arrhythmia • Pneumoperitoneum • PEEP ≥15 • RV failure
Backup Only
PLAX Fallback — When Subcostal EF is Equivocal
Only if your eyeball EF is borderline (you can't decide between mild and moderate). Try PLAX for EPSS or FS.
PLAX Probe Position
PLAX — Labeled
PLAX — Self-Test
CL PLAX Tips ⚠ DIFFICULT
Making PLAX Work on the Curvilinear
📐
Tilt on edge: Don't place the probe flat. Stand it up so only the center of the curved face contacts skin in the intercostal space. You're using ~20% of the array.
💧
Excessive gel: The tilted angle creates air gaps. Fill generously.
⬇️
Go lower: Try 4th–5th ICS instead of 3rd–4th. Wider spaces lower down.
↰
Left tilt: Under GA, 15° left table tilt. Awake, ask them to roll slightly left.
🎯
If you get a window: EPSS is fastest (M-mode through MV tip, measure E to septum). <7mm normal, >13mm severe.
EPSS: <7mm = Normal | 7–13mm = Reduced | >13mm = Severe
M-Mode MV — Anatomy
M-Mode EPSS — Measurement Example
📏
EPSS (E-Point Septal Separation): Place M-mode cursor through the tip of the anterior mitral leaflet. Measure the distance from the E-point (maximum opening) to the IVS. The E-point is the tall peak; the A-point is the smaller peak after it.
⚡
Why it works: In a healthy heart, the MV anterior leaflet swings wide and nearly touches the septum (small EPSS). In a failing heart, the LV is dilated and hypokinetic — the leaflet doesn't reach the septum (large EPSS).
⚠️
Pitfalls: EPSS is unreliable with significant AR, MS, or aortic root dilation — all push the leaflet away from the septum independent of EF.
EPSS Calculator (if you get a PLAX window)
EPSSmm
—
EPSS (mm)
—
Est. EF
Shock Recognition & Resuscitation
RUSH Protocol — HI-MAP Approach
Shock is a physiological emergency, not just low BP. Normal BP ≠ no shock. POCUS identifies life-threatening causes in seconds — treat while you diagnose.
Learning Objectives
✔️
Understand SHOCK as a physiological emergency — not just low BP.
✔️
Apply the RUSH Protocol using the HI-MAP approach: Heart → IVC → Morrison's → Aorta → Pneumothorax.
✔️
Use POCUS to identify life-threatening causes in seconds.
✔️
Apply ABCDE resuscitation with ultrasound integration.
Treat: Vasopressors (norepi), early antibiotics if septic, source control.
Key Ultrasound Findings — Pattern Recognition
🫀
Poor LV contractility → cardiogenic shock
💧
Collapsed IVC → hypovolemia
🫁
B-lines → pulmonary edema
🩸
Free fluid (FAST+) → internal bleeding
🚫
Absent lung sliding → pneumothorax
⚠ Avoid Deadly Mistakes in Early Shock
Anchoring on a normal BP. Giving fluids to a failing pump. Missing tamponade or tension PTX because you didn't look. Delaying pressors while chasing a diagnosis. Treat while you diagnose.
🧠 💥 High-Yield Teaching Pearls (Exam Gold)
🔑
Normal BP ≠ No shock. Compensated shock kills — look at lactate, mentation, cap refill, urine output.
🔑
Lactate = tissue hypoxia marker. Trend it — a falling lactate means your resuscitation is working.
🔑
IVC = your circulatory "dipstick". Quick, bedside, repeatable — but respect its limits (PEEP, pneumoperitoneum).
🔑
Treat while you diagnose — DO NOT WAIT. Pressors, blood, decompression happen in parallel with the scan.